Increases in nuclear p65 activation in dystrophic skeletal muscle are secondary to increases in the cellular expression of p65 and are not solely produced by increases in IκB-α kinase activity
Rajvir Singh, Gregory Millman, Eric Turin, Lucasz Polisiakeiwicz, Brian Lee, Francesca Gatti, Jonas Berge, Emily Smith, John Rutter, Chris Sumski, W. Tyler Winders, Abbas Samadi, C. George CarlsonVolume:
285
Year:
2009
Language:
english
DOI:
10.1016/j.jns.2009.06.030
File:
PDF, 1.57 MB
english, 2009