Targeted deletion of the antisilencer/enhancer (ASE) element from intron 1 of the myelin proteolipid protein gene ( Plp1 ) in mouse reveals that the element is dispensable for Plp1 expression in brain during development and remyelination
Pereira, Glauber B., Meng, Fanxue, Kockara, Neriman T., Yang, Baoli, Wight, Patricia A.Volume:
124
Language:
english
Journal:
Journal of Neurochemistry
DOI:
10.1111/jnc.12092
Date:
February, 2013
File:
PDF, 647 KB
english, 2013