The Local Environment at the Cytoplasmic End of TM6 of the μ Opioid Receptor Differs from Those of Rhodopsin and Monoamine Receptors:  Introduction of an Ionic Lock between the Cytoplasmic Ends of Helices 3 and 6 by a L6.30(275)E Mutation Inactivates the μ Opioid Receptor and Reduces the Constitutive Activity of Its T6.34(279)K Mutant †

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