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Increased levels of UCHL1 are a compensatory response to disrupted ubiquitin homeostasis in spinal muscular atrophy and do not represent a viable therapeutic target
Powis, Rachael A., Mutsaers, Chantal A., Wishart, Thomas. M., Hunter, Gillian, Wirth, Brunhilde, Gillingwater, Thomas H.Volume:
40
Language:
english
Journal:
Neuropathology and Applied Neurobiology
DOI:
10.1111/nan.12168
Date:
December, 2014
File:
PDF, 866 KB
english, 2014