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Defects in assembly explain reduced antiviral activity of the G249D polymorphism in human TRIM5α
Kömürlü, Sevnur, Bradley, Margret, Smolin, Nikolai, Imam, Sabrina, Pauszek, Raymond F., Robia, Seth L., Millar, David, Nakayama, Emi E., Shioda, Tatsuo, Campbell, Edward M., Menéndez-Arias, LuisVolume:
14
Language:
english
Journal:
PLOS ONE
DOI:
10.1371/journal.pone.0212888
Date:
March, 2019
File:
PDF, 2.21 MB
english, 2019